Activation of PI3K, Akt, and ERK during early rotavirus infection leads to V-ATPase-dependent endosomal acidification required for uncoating | PLOS Pathogens
Suppression of ERK phosphorylation through oxidative stress is involved in the mechanism underlying sevoflurane-induced toxicity in the developing brain | Scientific Reports
JCI - ERK1/2-Akt1 crosstalk regulates arteriogenesis in mice and zebrafish
Extracellular Regulated Kinases (ERK) 1 and ERK2 Are Authentic Substrates for the Dual-specificity Protein-tyrosine Phosphatase
MAPK ERK Signaling Regulates the TGF-β1-Dependent Mosquito Response to Plasmodium falciparum | PLOS Pathogens
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Western blot analysis of p/total‑Akt, p‑ERK/total‑ERK and cleaved... | Download Scientific Diagram
MAPK/ERK and JNK pathways regulate lipid synthesis and cell growth of Chlamydomonas reinhardtii under osmotic stress, respectively | Scientific Reports
Purified Mouse Anti-ERK (pan ERK)
Figure 4 | PLOS ONE
p-ERK Antibody (E-4) | SCBT - Santa Cruz Biotechnology
Phospho-ERK1/ERK2 (Thr202, Tyr204) Polyclonal Antibody (36-8800)
Western blot analysis for Erk 44/42MAPK phosphorylation in alpha cells (TC1-6 and InR1G9).
Western blot analysis for phospho-p38MAPK, phospho-ERK and phospho-Akt.
12-O-Tetradecanoylphorbol-13-acetate increases cardiomyogenesis through PKC/ ERK signaling - Section of Animal Physiology and Immunology
High Erk-1 activation and Gadd45a expression as prognostic markers in high risk pediatric haemolymphoproliferative diseases | Journal of Experimental & Clinical Cancer Research | Full Text
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ERK and Akt signaling pathways are involved in advanced glycation end product-induced autophagy in rat vascular smooth muscle cells
Extracellular Regulated Kinases (ERK) 1 and ERK2 Are Authentic Substrates for the Dual-specificity Protein-tyrosine Phosphatase
Phospho-ERK1/2 (Thr202/Tyr204) antibody (80031-1-RR) | Proteintech
STAT3 inhibitor sensitized KRAS-mutant lung cancers to RAF inhibitor by activating MEK/ERK signaling pathway - Figure f4 | Aging
IJMS | Free Full-Text | Nrf2-Mediated HO-1 Induction Coupled with the ERK Signaling Pathway Contributes to Indirect Antioxidant Capacity of Caffeic Acid Phenethyl Ester in HepG2 Cells
